Peritoneal inflammatory responses in the injured abdomen

  • Research type

    Research Study

  • Full title

    The Role of Intestinal Peritoneum and Biologic Implants in Inflammatory Responses and Healing in the Injured Abdomen

  • IRAS ID

    160798

  • Contact name

    James Berry

  • Contact email

    james.berry@manchester.ac.uk

  • Sponsor organisation

    University Of Manchester

  • Clinicaltrials.gov Identifier

    15/LO/0490, NRES Committee London - City Road Hampstead

  • Duration of Study in the UK

    1 years, 11 months, 26 days

  • Research summary

    Peritoneal adhesions are bands of scar tissue that form between abdominal organs and abdominal wall. They are common following trauma, infection and surgery and may cause significant morbidity, often requiring further surgical treatment. This surgery is especially challenging following multiple operations in which the abdominal wall is compromised and may require reinforcement or replacement by prosthetic implant material. Although such implanted material is said to be inert, certain types of biological implants have been shown to cause particularly severe adhesions.
    Peritoneal adhesions are a significant burden on the NHS and treating them is estimated to cost the NHS £67 million each year. Although adhesions seem to develop as a result of inflammation within the abdomen, the precise underlying mechanisms are not fully understood and there are still no reliable methods of adhesion prevention.
    The proposed studies examine the role of the peritoneal lining in the coordination of the inflammatory responses that lead to adhesion formation, to identify potential future targets for preventative strategies.
    The proposed studies will be undertaken in samples taken from patients undergoing elective abdominal surgery. Explanted peritoneal tissues will be cultured in the laboratory and exposed to a inflammatory stimulus from gut bacteria (lipopolysaccharide), as well as biological implants. Expression of key regulatory genes and proteins and the signalling pathways involved in the inflammatory response will be examined in these tissues.

  • REC name

    London - Bromley Research Ethics Committee

  • REC reference

    15/LO/0490

  • Date of REC Opinion

    24 Mar 2015

  • REC opinion

    Further Information Favourable Opinion