Glucagon in MODY

  • Research type

    Research Study

  • Full title

    Investigating glucagon secretion in HNF1-alpha and HNF4-alpha MODY

  • IRAS ID

    221972

  • Contact name

    Ioannis Spiliotis

  • Contact email

    ioannis.spiliotis@ocdem.ox.ac.uk

  • Sponsor organisation

    University of Oxford

  • Clinicaltrials.gov Identifier

    2016-005254-38, EudraCT

  • Duration of Study in the UK

    0 years, 6 months, 1 days

  • Research summary

    Diabetes is a chronic condition that affects 1 in 16 people in the UK, and leads to difficulty controlling blood sugar levels. This is due to an imbalance between two main hormones: insulin, which lowers blood sugar, and glucagon, which causes it to rise. Most patients have type 2 diabetes and are managed on a number of different medications including a new class called SGLT-2 inhibitors. MODY (Maturity Onset Diabetes of the Young) is a form of diabetes due to specific gene mutations and affects a much smaller group of patients. However, they are usually managed very well on one specific class of medication called sulphonylureas. Recent studies have suggested a link between MODY and the way SGLT-2 inhibitors work. The aim of this study is to better understand the role of glucagon in MODY patients, as this may point towards an improved treatment strategy of using SGLT-2 inhibitors along with low doses of sulphonylureas in type 2 diabetes.

  • REC name

    South Central - Hampshire B Research Ethics Committee

  • REC reference

    17/SC/0233

  • Date of REC Opinion

    5 May 2017

  • REC opinion

    Favourable Opinion