CANTOS:CACZ885M2301 Canakinumab in postMI patients with raised hsCRP

  • Research type

    Research Study

  • Full title

    A randomized, double-blind, placebo-controlled, event-driven trial of quarterly subcutaneous canakinumab in the prevention of recurrent cardiovascular events among stable post-myocardial infarction patients with elevated hsCRP

  • IRAS ID

    92727

  • Contact name

    Marcus Denis Flather

  • Contact email

    marcus.flather@uea.ac.uk

  • Sponsor organisation

    Novartis Pharmaceuticals UK Ltd

  • Eudract number

    2010-022970-14

  • Clinicaltrials.gov Identifier

    NCT01327846

  • Clinicaltrials.gov Identifier

    CCRN 738, NIHR adoption number

  • Duration of Study in the UK

    4 years, 6 months, 9 days

  • Research summary

    Myocardial infarction (or heart attack) is mainly caused by blockage of a coronary artery from a build up of fatty material and inflammatory cells called atherosclerosis. Atherosclerosis is a disease characterized by a high state of inflammation in the blood vessels.
    Certain inflammatory mediators called interleukins have been shown to be key mediators of this inflammation, in particular interleukin-1β (IL-1β), making it a reasonable target for reducing blood vessel inflammation associated with atherosclerosis. Canakinumab (ACZ885) is a fully human monoclonal anti-human IL-1β antibody, being developed for the treatment of IL-1β driven inflammatory diseases. High sensitivity C-reactive protein (hs-CRP) is a marker of ongoing inflammation that is readily measured by doing a blood test, and therefore patients with high levels of inflammation after heart attack can readily be identified for this study. In these patients, canakinumab is expected to reduce the risk of future occurrence of major cardiovascular events by preventing further blood vessel inflammation.
    Atherosclerosis is the primary cause of illness and death in individuals with and without type 2 diabetes mellitus (T2DM). Heart attack, stroke and blood vessel disease occur at higher frequency in T2DM patients and continue to increase despite use of current optimal therapies such as glucose-lowering, lipid-lowering, blood pressure lowering, and anti-clotting therapies.
    Canakinumab is expected to prevent new onset T2DM in patients with a recent past heart attack and who are at risk of developing T2DM. No comparative anti-inflammatory treatment is yet prescribed in patients with cardiovascular disease, and therefore the study is placebo-controlled on top of standard therapies, without an active comparator arm.
    The study duration for each participant will be up to approximately 6 years including 2 injections just below the skin at each visit (randomisation, Wk 2, Wk 12 then quarterly).
    About 200 patients will be randomised in the UK from approximately 26 sites.

  • REC name

    East Midlands - Leicester Central Research Ethics Committee

  • REC reference

    12/EM/0018

  • Date of REC Opinion

    15 Feb 2012

  • REC opinion

    Further Information Favourable Opinion